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TheInventor's Research Findings:
By
Frank Aidoo. Ph.D. Enzyme & Alkaloid Research Scientist.
In
his research work spanning over 20 years, Prof: Frank Aidoo, the first scientist
to discover and explain the different forms of alopecia unknown within the
scientific community, has also, by virtue of the proprietorship of his work,
ascribed names for their entry.
Enumerated
below, you will find for the first time, the cause of hair loss--what is
commonly referred to as alopecia--including its different forms.
Before
his discovery--and the breakthrough leading to the "re-activation" of
the dormant hair follicles to naturally grow hair again, science had no clue as
to the cause of hair loss. Many in the scientific community including physicians
within the define of dermatology, ascribed to lack of testosterone as the cause.
This, by its sphere of unfulfilled scientific data--an ascension of conjecture
was developed! By equation, and, as theory of scientific lack, different forms
of androgens have been tried to elicit hair growth without success; including
the use of minoxidil: a topical "drug" serendipitously hailed as a
"stimulant" to grow hair. Again, it has become known within the
scientific community that the prevailing chain of format within the base of this
topical does not suffice an answer to hair loss or alopecia.
For the
first time in human existence, an unequivocal exegesis of scientific standing
and wording--with testament of scientific data, all collected to substantiate
the tonation of its worth to humankind has been achieved. A breakthrough
unequaled--as it pertains to alopecia or hair loss has at last been achieved.
The
new wording and terminology listed below were coined by Prof: Aidoo to accent
their description.
(Please, note these names are copyright
of Aidoo Enzyme & Alkaloid Research Foundation).
1]
gHanape-bio
2] Tematitis A & D
3] Khalahali-vdex Hair
4] Bron-etitis
5] gEnecidal-lasidex Syndrome
6] Oxtiditronomidex Syndrome
7] Lint-lasidex Syndrome
gHanape-bio.
This is the name of the unnatural
membrane that develops to restrict the growth of the hair follicle. In my
research, I discovered that within the three stages of the hair state.
a) Anagen, b) Catagen and c) Telogen. A
state of telogenency becomes apparent when the natural composite of keratin and
sebum secretion earmarked for the upkeep of the hair follicles are utilized by
the gHanape-bio.
The use
of these natural composites help enrich and "prosper" the advent of
the suppresser--the gHanape-bio.
All
forms of alopecia are initiated by this exertion of dominance by the gHanape-bio.
Tematitis:
A & D
This is a form of Hair loss which
affect people of African descent. The composition of sufferers is: 80% female:
Tematitis D. and 20% male: Tematitis A. It usually starts from the hairline and
works its way toward the vertex area.
--Within
the define of Tematitis, the hair begins to break off to the very tip of the
scalp; remaining stagnant--in a "dwarftanian" fashion.
Khalahali--vdex
hair
Sufferers have what I have named:
"Khalahali--vdex hair". ...It is the most driest type of human hair,
with very tight curls. It is also the most efficient user of the human sebumatic
secretion (sebum); using a third of this natural secretion--compared to other
bearers of sub-African ancestry.
By its
genetic composition, the Khalahali-vdex hair is very light in weight among all
other human hair... The cuticle bonds present within the Khalahali-vdex hair
type are the same as the other group of inheritors of the African ancestral
hair. It is also the type of hair whose epic of density--including length,
seldom exceeds the hairline when a strand in the vertex area is elasticated
toward the side of the head.
The
syndrome of Tematitis is established within 4 to 5 weeks after the first cycle
of hair loss. The follicles become entombed within the gHanape-bio outlay, as
the emitting of the remaining hairfiber lying between the epidermis and the
dermis--outside the perimeters of the gHanape-bio track--within the shaft--is
expelled out due to its lack of sustenance from the follicle.
The
syndrome known scientifically as catagenency then becomes evident.
The
follicle, now under this debased state of pre-eminence, is deduced into semi
"starved" state, as the gHanape-bio proceeds to create layers of
"cocoon over cocoon" --restricting all sebumatic secretion from
reaching it.
After a
period of another 5 weeks at most, the scalp begins to show telogenic
characteristics--giving out "oily" appearance.
Bron-etitis

This form of hair oss affects people of
the Caucasian race. It is what Tematitis is to people of African descent. The
only difference being: Bron-etitis is suffered extensively by both male and
female within the Caucasian race. The hair loss "patches" do not start
from the sides of the head like Tematitis. A sufferer wakes up to find hair loss
in patches with no apparent warning. The hair loss continues unabated, with
every passing day's loss becoming heavier than before.
gEnicidal-lasidex
Syndrome
My research has shown that, due to the
lower density of the cuticle bonds within this type of hair, a syndrome I have
named: gEnicidal-lasidex sets in. This is where the follicles of the sufferer
mounts a monotratic accord, conceding to the presence of the "lead"
gHanape-bio attack... The first follicle attacked relay this intrusion to the
others around its sphere, resulting in a frantic syndrome of
discomfort--creating a progressive denaturing sway of imbalance; and allowing
the gHanape-bio to take advantage of the situation. ...A large portion of the
hair-follicles within this region succumb to this define!
...
Most
physicians result to the use of androgens; usually topicals such as cortisone.
My work
has shown that the use of such drugs usually presents side effects--when
syringecally administered; --epidermal contortion of the injected area
results... Infact, when such aggressive counter "bluff" is exerted
over the formed areas, there is always the danger of awarding the gHanape-bio
extra "territory!" --Territory not yet under its domain.
My
research has also shown that testosterone vestiges as combactors are only
temporal--even in the most encouraging way... For their chemical composition and
molecular format tend to denature the other sebumatic secretions not yet
entwined within the element of the conquering gHanape-bio.
The use
of AidooAid with its proportioned counter balanced enzymatic precepts of protein
and alkaloidic properties, including the prevailing nitrogenous
composites--arranged as co-factors of import, charge and catalyze endergonically--within
the same density of equilibrium; hence, effecting an accord to counteract the
outlay of destructiveness coherent within the gHanape-bio's traits.
Some
properiants within the composite of the "follicle re-activator," the
final phase of the AidooAid System, are actually proteinic semblance of the
follicle's keratinic needs. And as such, when used as directed, help to
naturally debase the gHanape-bio's grip.
...Being
physiologically semblance to the human secretion of sebum and all other
providers of the bulbing needs, it redresses all ineptitude brought about by
this unnatural swadex; as it fosters a natural recovery from the entangled
swipes of this syndrome.
Oxtiditronomidex
Syndrome
This is a form of Alopecia which affect
the male population within all ethnicity and race.
Within
the define of Oxtiditronomidex Syndrome, the hair loss becomes conspicuous in
two places: the "vertex and frontal" areas. Usually, the vertex patch
or area of hair loss is twice the size of the frontal. The result here is that,
if not treated, the "bridge" (this is the hair lying between the
Ostiditronomidex--the two patches of hair loss) eventually join together;
leaving an "Avocado" shaped single patch of baldness.
Lint-lasidex
Syndrome
This type of Alopecia affects both male
and female. It always starts from the back of the head--the hairline above the
neck.
...Again,
it begins when the composite of the sebumatic secretion into the hair
follicles--over the area covering the head is utilized by the gHanape-bio. The
sufferer begins to notice that his shirt collar--behind his head, falls very low
from the hairline.
In the
case of female, this syndrome begins by extending both the front and back
hairline. It eventually leaves a longer-prominent forehead, while at the same
time moving the hairline above the neck inches from its original mark.
...On
the sides of the head--above the ears, one will also notice an abnormal gap
between the hairline and the ears; and a semi-horizontal hairline above the
ears.
The
atomic make up of the base in the AidooAid
"Follicle Activator"product is calculated to fire its nuclei composite within an
equilibrium of formatting--achieving therefore a near zero cellosteric
compounding.
--There
is an accentuation of 0/2 denaturing superfluous within this firing.
The
content of this page is copyrighted. Used with Permission. All Right Reserved.
The pictorials we show are various customers'
testimonials for men and women from all races who have used The AidooAid® Hair
Follicle Re-Activation System™ and provided their individual experiences. The product is not intended to treat or prevent
disease or grow hair or otherwise affect the structure or functions of the human
body.
AidooAid: Hair Follicle Re-Activation System, AidooAid
MaintyDex System, AidooAid Hair Loss Combator, AidooAid The Bald No More System.
AidooAid Ancestral:are Registered Trademarks of Aidoo Enzyme and Alkaloids
Research. [Aidoo Ancestral Products Inc., Aidoo International Research &
Marketing Inc.] Copyright 1996-2001. All Rights Reserved.
The use and copying of any picture/data is subject to License
Agreement. Any other use is prohibited: No part of it may be reproduced,
transmitted, transcribed, stored in a retrieval system or translated into any
language in any form by any means without the prior written consent of Aidoo
International Research & Marketing Inc., Loma Linda, California, USA.
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